Prognostic value of cardiovascular biomarkers in patients with suspected coronary artery disease

https://doi.org/10.1007/s00392-025-02625-4

Beya Abdennadher (Hamburg)1, A. Goßling (Hamburg)1, J. Koschwitz (Hamburg)1, L. Paasch (Hamburg)1, N. A. Sörensen (Hamburg)2, T. Zeller (Hamburg)3, R. Twerenbold (Hamburg)3, S. Blankenberg (Hamburg)3, C. Sinning (Hamburg)2, J. T. Neumann (Hamburg)4

1Universitätsklinikum Hamburg-Eppendorf Klinik für Kardiologie Hamburg, Deutschland; 2Universitäres Herz- und Gefäßzentrum Hamburg Klinik und Poliklinik für Kardiologie Hamburg, Deutschland; 3Universitäres Herz- und Gefäßzentrum Hamburg Klinik für Kardiologie Hamburg, Deutschland; 4Universitäres Herz- und Gefäßzentrum Hamburg Allgemeine und Interventionelle Kardiologie Hamburg, Deutschland

 

Methods: Patients undergoing cardiac stress testing for suspected coronary artery dis-ease were recruited in a prospective cohort study. Measurements of high-sensitivity cardiac troponin (hs-cTnI), high-sensitivity C-reactive protein (hs-CRP), and natriuretic peptides (NT-proBNP) were obtained before the stress test. Hs-cTnI concentrations were also measured one hour after the stress test. The diagnostic performance of each biomarker in predicting a pathological stress test was evaluated by calculating the area under the receiver operating characteristic curve. Cox regression analysis was conduct-ed to identify predictors of incident major adverse cardiovascular events (MACE).

Results: 778 participants (median age 70 years) were included in the final analysis, of which 240 had a pathological stress test and 558 a non-pathological stress test. During follow-up of 33 months (IQR 32-34), an event rate of 17.8 % was reported for MACE. Baseline hs-cTnI and NT-proBNP concentrations were significantly higher in participants with a pathological stress test (8.0 ng/L vs. 4.5 ng/L and 398.9 vs. 261.6 ng/L, respective-ly). Hs-cTnI concentrations after one hour showed a slight decrease from the baseline concentration. 
All biomarker concentrations showed poor performance in discriminating between pathological and non-pathological stress tests. Concentrations of hs-cTnI and NT-proBNP were significantly associated with incident MACE after adjusting for conventional cardiovascular risk factors (HR 1.29 per SD [95% CI 1.15, 1.45] and 1.19 per SD [95%CI 1.07, 1.32] respectively)

Conclusion: In patients undergoing cardiac stress testing, concentrations of hs-cTnI, NT-proBNP, and hs-CRP demonstrated limited ability in discriminating between pathological and non-pathological test results. However, hs-cTnI and NT-proBNP concentrations were predictors of incident MACE beyond established cardiovascular risk factors sug-gesting their potential to improve cardiovascular risk assessment in high-risk popula-tions.
 
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