Impact of left ventricular dysfunction on outcomes in microaxial flow-pump-supported high-risk PCI

https://doi.org/10.1007/s00392-025-02737-x

Alexander Laupp (Dresden)1, F. Woitek (Dresden)1, A. Conrad (Dresden)1, S. Haußig (Dresden)1, J. Mierke (Dresden)1, J. Vogel (Dresden)2, E. B. Winzer (Dresden)1, R. Höllriegel (Dresden)1, S. Jellinghaus (Dresden)1, A. Linke (Dresden)3, N. Mangner (Dresden)1

1Herzzentrum Dresden GmbH an der TU Dresden Klinik für Innere Medizin, Kardiologie und Intensivmedizin Dresden, Deutschland; 2Herzzentrum Dresden GmbH an der TU Dresden Dresden, Deutschland; 3Herzzentrum Dresden GmbH an der TU Dresden Klinik für Innere Medizin und Kardiologie Dresden, Deutschland

 

 

Background: Patients undergoing high-risk percutaneous coronary intervention (HRPCI) are characterized by complex coronary artery disease, severe comorbidities, and adverse hemodynamics, including reduced left ventricular ejection fraction (LV-EF). We investigated the optimal cut-off of LV-EF in patients undergoing microaxial flow pump (mAFP)-supported HRPCI to predict survival, clinical functional outcomes, and a PROTECT IV-like composite endpoint, and aimed to evaluate the impact of complete revascularization in patients with LV-EF≤40% versus LV-EF>40%.


Methods:
 We used a prospective single-center registry including 225 patients undergoing mAFP-supported HRPCI. Receiver operating curve analysis determined the optimal LV-EF cut-off for predicting 3-year survival. The primary endpoint was 3-year survival; safety was assessed by ARC-2 and VARC-3 definitions. Secondary endpoints included PROTECT IV-like composite outcomes and functional status (NYHA, CCS, LV-EF). The interaction between complete revascularization and LV-EF status was analyzed.


Results:
 Receiver operating curve analysis identified an LV-EF cut-off of 40% for predicting 3-year survival. Patients were stratified into LV-EF≤40% (n=123) and LV-EF>40% (n=102) groups. LV-EF≤40% patients were younger, exhibited more severe heart failure symptoms, and had higher STS scores (median 7.0% vs. 4.3%, p<0.001). The prevalence of 3-vessel disease (~80%), baseline SYNTAX score (32 (IQR 24; 40), and BCIS Jeopardy Score (12 (IQR 8; 12) were high in both groups without significant differences. However, the rate of any CTO was higher, and left main disease was less frequent in LV-EF≤40%.

Complete revascularization (residual BCIS Jeopardy Score <4) was numerically less frequent in LV-EF≤40% (69.1% vs. 76.5%, p=0.218). Duration of mAFP support was comparable. Major access-site complications occurred more often in the LV-EF>40% group (26.5% vs. 13.1%, p=0.011), while rates of myocardial infarction, stent thrombosis, stroke, acute kidney injury, and bleeding were similar.

Three-year survival was significantly lower in LV-EF≤40% (53.7% vs. 70.6%, p=0.002). In multivariate Cox regression analysis, LV-EF≤40% was independently associated with increased 3-year mortality (HR 1.76; 95% CI 1.11-2.81). Complete revascularization had no survival benefit in LV-EF>40% (68.4% vs. 73.9%, p=0.556) but was associated with improved survival in LV-EF≤40% (59.5% vs. 39.5%, p=0.014; p-value for interaction <0.001).

At a median follow-up of 977 (IQR 426–1826) days, NYHA and CCS class III/IV symptoms were reduced by 68.5% and 75.2%, respectively. Reverse LV remodeling was more pronounced in LV-EF≤40% (10.0% vs. 0.5%, p<0.001). The PROTECT IV-like composite endpoint at 3 years favoured LV-EF>40% (54.9% vs. 45.5%, p=0.025).


Conclusion:
 In patients undergoing mAFP-supported HRPCI, a LV-EF≤40% was associated with poor 3-year survival. Complete revascularization led to a higher 3-year survival in patients with LV-EF≤40%, but had no impact on patients with LV-EF>40%. The LV-EF≤40% population appears suitable for studying the effects of a mAFP-guided HRPCI strategy aimed at achieving complete revascularization in randomized controlled trials, such as the PROTECT IV study.

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