Mechanisms of Atrial Fibrillation

Donnerstag, 4. April | 14:30 - 16:00 Uhr | Posterbereich 28

Vorsitz/Chair: Sebastian Clauß (München), Markus Hecker (Heidelberg)


Does The Fractionation of Exposure to Nanosecond Pulsed Electric Fields Matter?
P. Sowa (Tübingen)
Expression of the potassium channel TASK-1 is regulated by the transcription factor ETV1 leading to changes in cardiac action potential morphology
M. Beck (Heidelberg)
Short and long-term antiarrhythmic effects of SGLT2i in a translational porcine large animal model
V. Herlt (Heidelberg)
Established Agents, New Mechanisms And Indications – Dapagliflozin Can Exert an Atrial-Predominant Class I Antiarrhythmic Effect
A. Paasche (Heidelberg)
Decreased NCX1 expression reduces proarrhythmogenic spontaneous Ca2+ releases in atrial cardiomyocytes from CREM-IbΔC-X transgenic mice, a model of atrial fibrillation
M. Gieske (Münster)
Novel high-density microelectrode array-based system for modelling human atrial re-entrant arrhythmias in Brugada syndrome
W. Li (Dresden)
Atrial Remodeling in Right Ventricular Pacing-induced Cardiomyopathy
M. Paulus (Regensburg)
Piezo1 as a Novel Link Between Fibrosis and Inflammation in Atrial Fibrillation
R. Emig (Freiburg im Breisgau)
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